Search results for "Zinc Fingers"

showing 10 items of 21 documents

Evolutionary diversification of type-2 HDAC structure, function and regulation in Nicotiana tabacum

2018

Ministère de l'Education Nationale et de la Recherche ; Conseil Régional de Bourgogne (PARI AGRALE8) ; Association pour la Recherche sur les Nicotianacées ; Conseil Régional de Bourgogne; International audience; Type-2 HDACs (HD2s) are plant-specific histone deacetylases that play diverse roles during development and in responses to biotic and abiotic stresses. In this study we characterized the six tobacco genes encoding HD2s that mainly differ by the presence or the absence of a typical zinc finger in their C-terminal part. Of particular interest, these HD2 genes exhibit a highly conserved intron/exon structure. We then further investigated the phylogenetic relationships among the HD2 gen…

0106 biological sciences0301 basic medicineNicotiana tabacumPlant Science01 natural sciencesEvolution MolecularType-2 HDAC (HD2)03 medical and health sciencesPhylogeneticsZinc fingerTobaccoGeneticsArabidopsis thalianaGene family[SDV.BV]Life Sciences [q-bio]/Vegetal BiologyAmino Acid SequenceGenePhylogenySolanaceaePlant ProteinsZinc fingerGeneticsbiologyModels GeneticIntronZinc FingersGeneral MedicineSalt Tolerancebiology.organism_classificationSalt stress responseComplementation030104 developmental biologyHistone DeACetylase (HDAC)Agronomy and Crop ScienceSequence Alignment010606 plant biology & botany
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Genotypic and phenotypic spectrum in tricho-rhino-phalangeal syndrome types I and III

2000

Tricho-rhino-phalangeal syndrome (TRPS) is characterized by craniofacial and skeletal abnormalities. Three subtypes have been described: TRPS I, caused by mutations in the TRPS1 gene on chromosome 8; TRPS II, a microdeletion syndrome affecting the TRPS1 and EXT1 genes; and TRPS III, a form with severe brachydactyly, due to short metacarpals, and severe short stature, but without exostoses. To investigate whether TRPS III is caused by TRPS1 mutations and to establish a genotype-phenotype correlation in TRPS, we performed extensive mutation analysis and evaluated the height and degree of brachydactyly in patients with TRPS I or TRPS III. We found 35 different mutations in 44 of 51 unrelated p…

AdultMaleanimal structuresAdolescentGenotypeDNA Mutational AnalysisMolecular Sequence DataLimb Deformities CongenitalBiologyOsteochondrodysplasiasPolymorphism Single NucleotideShort statureLanger–Giedion syndromeGeneticsmedicineHumansMissense mutationTricho–rhino–phalangeal syndromeGenetics(clinical)Amino Acid SequenceChildGenetics (clinical)GeneticsAnthropometryBase SequenceBrachydactylyInfantZinc FingersExonsSyndromeArticlesMiddle AgedMicrodeletion syndromemedicine.diseasePenetranceBody HeightPedigreeDNA-Binding ProteinsRadiographyPhenotypeChild PreschoolMutationTrichorhinophalangeal Syndrome Type IErythroid-Specific DNA-Binding FactorsFemalemedicine.symptomChromosomes Human Pair 8Transcription Factors
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Negative Regulation of β Enolase Gene Transcription in Embryonic Muscle Is Dependent upon a Zinc Finger Factor That Binds to the G-rich Box within th…

1998

We have previously identified a muscle-specific enhancer within the first intron of the human beta enolase gene. Present in this enhancer are an A/T-rich box that binds MEF-2 protein(s) and a G-rich box (AGTGGGGGAGGGGGCTGCG) that interacts with ubiquitously expressed factors. Both elements are required for tissue-specific expression of the gene in skeletal muscle cells. Here, we report the identification and characterization of a Kruppel-like zinc finger protein, termed beta enolase repressor factor 1, that binds in a sequence-specific manner to the G-rich box and functions as a repressor of the beta enolase gene transcription in transient transfection assays. Using fusion polypeptides of b…

AgingTranscription GeneticMolecular Sequence DataDown-RegulationRepressorRegulatory Sequences Nucleic AcidBiologyBiochemistryDNA-binding proteinGene Expression Regulation EnzymologicMiceGene expressionAnimalsHumansAmino Acid SequenceCloning MolecularMuscle SkeletalEnhancerMolecular BiologyCell NucleusRegulation of gene expressionZinc fingerSp1 transcription factorBinding SitesSequence Homology Amino AcidZinc FingersCell BiologyMolecular biologyDNA-Binding ProteinsEnhancer Elements GeneticRegulatory sequencePhosphopyruvate HydrataseJournal of Biological Chemistry
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Specific bovine antibody response against a new recombinant Cryptosporidium parvum antigen containing 4 zinc-finger motifs

2002

A Cryptosporidium parvum sporozoite and oocyst lambda gt11 cDNA library was screened with a hyperimmune rabbit serum that was developed against insoluble fragments of ultrasonicated oocysts. A clone named Cp22.4.1 encoding a protein of 231 amino acids with 4 zinc-finger domains characterized by a Cys-X2-Cys-X4-His-X4-Cys motif was isolated and characterized. There was a complete match between the sequencing data of the coding region of Cp22.4.1 and the corresponding gene at chromosomal level. Cloning in a pBAD-TOPO-TA expression vector permitted to evaluate the antigenicity of the recombinant His-tagged antigen. This antigen was recognized by 2 out of 5 sera from Cryptosporidium immune calv…

Antigenicityanimal diseasesMolecular Sequence DataProtozoan ProteinsAntibodies ProtozoanAntigens ProtozoanMolecular cloningBrief Communicationlaw.inventionAntigenlawparasitic diseasesAnimalsAmino Acid SequenceZinc fingerCryptosporidium parvumExpression vectorbiologyBase SequencecDNA libraryZinc Fingersbiology.organism_classificationVirologyRecombinant ProteinsInfectious DiseasesCryptosporidium parvumRecombinant DNAParasitologyCattleRabbits
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The Muscleblind family of proteins: an emerging class of regulators of developmentally programmed alternative splicing.

2006

Alternative splicing is widely used to generate protein diversity and to control gene expression in many biological processes, including cell fate determination and apoptosis. In this review, we focus on the Muscleblind family of tissue-specific alternative splicing regulators. Muscleblind proteins bind pre-mRNA through an evolutionarily conserved tandem CCCH zinc finger domain. Human Muscleblind homologs MBNL1, MBNL2 and MBNL3 promote inclusion or exclusion of specific exons on different pre-mRNAs by antagonizing the activity of CUG-BP and ETR-3-like factors (CELF proteins) bound to distinct intronic sites. The relative activities of Muscleblind and CELF proteins control a key developmenta…

Cancer ResearchCellular differentiationMolecular Sequence DataRNA-binding proteinCell fate determinationBiologychemistry.chemical_compoundExonMiceMBNL1AnimalsHumansMyotonic DystrophyAmino Acid SequenceMolecular BiologyGeneticsZinc fingerAlternative splicingGene Expression Regulation DevelopmentalRNA-Binding ProteinsCell DifferentiationZinc FingersCell BiologyAlternative SplicingchemistryRNA splicingDevelopmental BiologyDifferentiation; research in biological diversity
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The pioneering role of PRDM9 indel mutations in tarsier evolution

2016

PRDM9 is currently the sole speciation gene found in vertebrates causing hybrid sterility probably due to incompatible alleles. Its role in defining the double strand break loci during the meiotic prophase I is crucial for proper chromosome segregation. Therefore, the rapid turnover of the loci determining zinc finger array seems to be causative for incompatibilities. We here investigated the zinc finger domain-containing exon of PRDM9 in 23 tarsiers. Tarsiers, the most basal extant haplorhine primates, exhibit two frameshifting indels at the 5'-end of the array. The first mutation event interrupts the reading frame and function while the second compensates both. The fixation of this allele…

Evolution MolecularINDEL MutationProtein DomainsTarsiidaeAnimalsZinc FingersHistone-Lysine N-MethyltransferaseArticle570 Biowissenschaften570 Life sciencesScientific Reports
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Gene Repair of an Usher Syndrome Causing Mutation by Zinc-Finger Nuclease Mediated Homologous Recombination

2012

PURPOSE. Human Usher syndrome (USH) is the most frequent cause of inherited deaf-blindness. It is clinically and genetically heterogeneous, assigned to three clinical types of which the most severe type is USH1. No effective treatment for the ophthalmic component of USH exists. Gene augmentation is an attractive strategy for hereditary retinal diseases. However, several USH genes, like USH1C, are expressed in various isoforms, hampering gene augmentation. As an alternative treatment strategy, we applied the zinc-finger nuclease (ZFN) technology for targeted gene repair of an USH1C, causing mutation by homologous recombination. METHODS. We designed ZFNs customized for the p.R31X nonsense mut…

Gene isoformNonsense mutationCell Cycle ProteinsBiologyRetinaCell Linechemistry.chemical_compoundHumansDNA Breaks Double-StrandedDNA CleavageHomologous RecombinationGeneAdaptor Proteins Signal TransducingZinc fingerGeneticsTargeted Gene RepairfungiZinc FingersDNAEndonucleasesZinc finger nucleaseCytoskeletal ProteinschemistryCodon NonsenseHomologous recombinationUsher SyndromesDNATargeted Gene RepairInvestigative Opthalmology & Visual Science
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The gene encoding the transcriptional repressor BERF-1 maps to a region of conserved synteny on mouse chromosome 16 and human chromosome 3 and a rela…

1999

We have recently identified and characterized a Kruppel-like zinc finger protein (BERF-1), that functions as a repressor of β enolase gene transcription. By interspecific backcross analysis the gene encoding BERF-1 was localized 4.7 cM proximal to the <i>Mtv6</i> locus on mouse chromosome 16, and an isolated pseudogene was localized to mouse chromosome 8, about 5.3 cM distal to the D8Mit4 marker. Nucleotide sequence identity and chomosome location indicate that the gene encoding BERF-1 is the mouse homologue (<i>Zfp148</i>) of ZNF148 localized to human chromosome 3q21, a common translocation site in acute myeloid leukemia patients.

Genetic MarkersDNA ComplementaryTranscription GeneticKruppel-Like Transcription FactorsBiologyHybrid CellsPolymerase Chain ReactionGene Expression Regulation EnzymologicMiceChromosome 16GeneticsAnimalsHumansMolecular BiologyGenetics (clinical)Conserved SequenceSyntenyDNA PrimersGeneticsBase SequenceYY1Chromosome MappingTAF9Zinc FingersTCF4DNA-Binding ProteinsRepressor ProteinsChromosome 3GATAD2BPhosphopyruvate Hydratasecardiovascular systemChromosomes Human Pair 3Chromosome 22PseudogenesTranscription FactorsCytogenetics and cell genetics
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The Saccharomyces cerevisiae zinc finger proteins Msn2p and Msn4p are required for transcriptional induction through the stress response element (STR…

1996

The MSN2 and MSN4 genes encode homologous and functionally redundant Cys2His2 zinc finger proteins. A disruption of both MSN2 and MSN4 genes results in a higher sensitivity to different stresses, including carbon source starvation, heat shock and severe osmotic and oxidative stresses. We show that MSN2 and MSN4 are required for activation of several yeast genes such as CTT1, DDR2 and HSP12, whose induction is mediated through stress-response elements (STREs). Msn2p and Msn4p are important factors for the stress-induced activation of STRE dependent promoters and bind specifically to STRE-containing oligonucleotides. Our results suggest that MSN2 and MSN4 encode a DNA-binding component of the…

Hot TemperatureSaccharomyces cerevisiae ProteinsTranscription GeneticSaccharomyces cerevisiaeMolecular Sequence DataPlasma protein bindingSaccharomyces cerevisiaeGeneral Biochemistry Genetics and Molecular BiologyTranscription (biology)Osmotic PressureMolecular BiologyGeneTranscription factorZinc fingerGeneticsGeneral Immunology and MicrobiologybiologyBase SequenceGeneral NeurosciencePromoterZinc Fingersbiology.organism_classificationYeastCell biologyDNA-Binding ProteinsOxidative StressOligodeoxyribonucleotidesResearch ArticleProtein BindingTranscription Factors
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Tristetraprolin Regulates the Expression of the Human Inducible Nitric-Oxide Synthase Gene

2005

The expression of human inducible NO synthase (iNOS) is regulated both by transcriptional and post-transcriptional mechanisms. Stabilization of mRNAs often depends on activation of p38 mitogen-activated protein kinase (p38 MAPK). In human DLD-1 cells, inhibition of p38 MAPK by the compound 4-(4-fluorophenyl)-2-(4-methylsulfinylphenyl)-5-(4-pyridyl)1H-imidazole (SB203580) or by overexpression of a dominant-negative p38 MAPKalpha protein resulted in a reduction of human iNOS mRNA and protein expression, whereas human iNOS promoter activity was not affected. An important RNA binding protein regulated by the p38 MAPK pathway and involved in the regulation of the stability of several mRNAs is tr…

ImmunoprecipitationRNA Stabilityp38 mitogen-activated protein kinasesTristetraprolinNitric Oxide Synthase Type IIRNA-binding proteinGene Expression Regulation EnzymologicCell LineImmediate-Early ProteinsTristetraprolinEnzyme StabilityHumansRNA MessengerProtein kinase APharmacologyRegulation of gene expressionbiologyChemistryZinc FingersTransfectionMolecular biologyDNA-Binding ProteinsNitric oxide synthasebiology.proteinMolecular MedicineNitric Oxide SynthaseMolecular Pharmacology
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